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Allergy"},"provenance":null},{"subject":{"scheme":"keyword","value":"Hepatitis A Virus Cellular Receptor 2"},"provenance":null},{"subject":{"scheme":"keyword","value":"Biomarkers"},"provenance":null},{"subject":{"scheme":"keyword","value":"post-covid condition"},"provenance":null},{"subject":{"scheme":"keyword","value":"Aged"},"provenance":null}],"mainTitle":"Markers of T cell activation and exhaustion in plasma are associated with persistent symptoms up to 18 months following mild SARS-CoV-2 infection","subTitle":null,"descriptions":["<jats:sec><jats:title>Background</jats:title><jats:p>Persistent symptoms following SARS-CoV-2 is an increasing problem after COVID-19 disease. The pathogenesis of this persistent post Covid-19 Condition (PCC) is, however, largely unknown. We hypothesized that persistent T cell activation and exhaustion play a role in PCC development.</jats:p></jats:sec><jats:sec><jats:title>Methods</jats:title><jats:p>We examined plasma levels of soluble (s) CD25, TIM-3 and LAG-3, all markers of T cell activation/exhaustion, by enzyme immunoassays in 170 home-isolated and 53 hospitalized patients for up to 18 months after COVID-19 in relation to persistent symptomatology.</jats:p></jats:sec><jats:sec><jats:title>Results</jats:title><jats:p>Our major findings were: (i) Cases with persistent dyspnea and fatigue had markedly higher sCD25 at 6–18 months with a more modest increase in sTIM-3. (ii) Cases with memory problems at 12–18 months had increased sLAG-3 iii) sCD25 correlated with SARS-CoV-2 antibody titers and microneutralization titers only in cases with PCC while sTIM-3 correlated with these parameters irrespectively of symptoms. iv) Although hospitalized patients had markedly elevated levels of T cell activation/exhausting markers during follow-up, there was no relation to PCC symptoms.</jats:p></jats:sec><jats:sec><jats:title>Conclusion</jats:title><jats:p>Our study indicates a role for T cell activation/exhaustion in PCC following home isolated COVID-19 infection, with somewhat different patterns of sCD25, sTIM-3 and sLAG-3, but not in hospitalized COVID-19 patients where disease severity may be more important.</jats:p></jats:sec>"],"publicationDate":"2025-05-30","publisher":"Frontiers Media SA","embargoEndDate":null,"sources":["Crossref","Front Immunol","Frontiers in Immunology, Vol 16 (2025)","Frontiers in 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